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Low mutation burden and frequent loss of CDKN2A/B and SMARCA2, but not PRC2, define premalignant neurofibromatosis type 1-associated atypical neurofibromas
LPL, synthesized in adipocytes, skeletal myocytes, and cardiomyocytes, is an essential enzyme for the lipolytic processing of TG in chylomicron and VLDL
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